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Criteria and Chaos: Diagnosing Fibromyalgia

  [ 19 votes ]   [ 6 Comments ]
By Celeste Cooper • • May 2, 2017

Criteria and Chaos: Diagnosing Fibromyalgia
According to the Centers for Disease Control and Prevention (CDC), approximately two percent of the US population is affected by fibromyalgia and the ratio of women to men is 7:1. But, are these statistics accurate?
Study design and the use of scientifically approved tools for measuring results is the benchmark for research. But, most important is an accurately defined study participant pool. First of its kind research using a community-based survey of 3,410 potential patients and a retrospective review of medical records of potential cases suggests patients, particularly men, are unlikely to be diagnosed with fibromyalgia.
The 2016 Revisions to the 2010/2011 Fibromyalgia Diagnostic Criteria appear to address previous concerns, and now conclude:
“This revision combines physician and questionnaire criteria, minimizes misclassification of regional pain disorders, and eliminates the previously confusing recommendation regarding diagnostic exclusions… The self-report version of the criteria is not valid for clinical diagnosis in individual patients but is valid for research studies.”
However, some doctors still rely on the tender point model as does Social Security,  see disability ruling SSR 12-2p: Titles II and XVI: Evaluation of Fibromyalgia. There are good arguments for doing away with the tender point count, and there are good arguments for keeping them. I suppose that is why the ACR does not completely exclude the tender point examination in their recommendation. The tender point count was the criteria when I was diagnosed. And, my FM/a® test results were astoundingly high, so for me—the criteria worked. But, does the tender point count work in diagnosing men? The consensus on this varies, too. Another disparity—few fibromyalgia studies include male subjects.
The FM/a® test will be used to identify fibromyalgia study participants for the genome study, just announced. So, there is hope and we are moving forward. I believe that if we can consistently identify patients using this blood test, and scientists are able to replicate the findings, we make a quantum leap for future research and a better understanding of fibromyalgia as a disease. To say that I am excited about this research is undeniable. I did have the FM/a test, I was positive, and I will be participating in the genome study.
Most agree there are four recurring characteristics: 
  • Body-wide pain and/or tenderness (The American College of Rheumatology [ACR], suggests assessment for a total of 19 painful areas on the body and their severity.)  
  • Dysfunctional, non-restorative sleep
  • Cognitive disturbance (fibro fog)
  • Fatigue
Fibromyalgia comorbid disorders - disorders that co-occur more frequently with fibromyalgia than with other diseases or syndromes
Comorbid disorders or signs (Resources: CDC, NIH, and the ACR. (Accessed, April 14, 2017)
  • Headache, severe, chronic*
  • Morning stiffness*
  • Tingling or numbness in hands and feet*
  • Depression or anxiety
  • Digestive problems, such as abdominal pain, bloating, constipation, irritable bowel syndrome (IBS), or gastroesophageal reflux disease (GERD)*
  • Pelvic pain, including irregularities with menstruation*
  • Irritable or overactive bladder*
  • Restless legs syndrome*
  • Sensitivity to loud noises or bright lights
  • Temperature sensitivity
  • Temporomandibular joint dysfunction (TMJ/TMD)* 
*Conditions that could be related to myofascial pain syndrome (MPS). This is important because there are specific, hands-on, treatments for MPS.
The CDC, National Institutes of Health (NIH), and ACR agree that we are more likely to develop fibromyalgia if a rheumatic disease such as osteoarthritis, lupus, rheumatoid arthritis, or ankylosing spondylitis is present. The presence of other painful disorders is a usual finding as noted in my review, The 2013 Fibromyalgia Alternative Diagnostic Criteria: Interpretation for Patients and Providers.

Though there are studies that have varying hypotheses, some proven, some not, there are factors in diagnosing fibromyalgia that are quite consistent. Because of this— fibromyalgia should NOT be a diagnosis of last resort.
Relating symptoms
It’s important for our doctor to know what they are treating and how we relate our symptoms to our physician can make a difference in getting the right diagnosis. The bottom line is if something is nagging at you, share it with your physician. You can find suggestions on how to do that here.
“Persistence and resilience only come from having been
given the chance to work though difficult problems.”
~Gever Tulley

Celeste Cooper, RN, is a frequent contributor to ProHealth.  She is an advocate, writer and published author, and a person living with chronic pain. Celeste is lead author of Integrative Therapies for Fibromyalgia, Chronic Fatigue Syndrome, and Myofascial Pain and Broken Body, Wounded Spirit, and Balancing the See Saw of Chronic Pain (a four book series). She spends her time enjoying her family and the rewards she receives from interacting with nature through her writing and photography. You can learn more about Celeste’s writing, advocacy work, helpful tips, and social network connections at

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Treating fibromyalgia
Posted by: meplatt1
May 2, 2017
The best way of treating fibromyalgia is to treat the underlying cause of this condition. Interestingly, doctors are rarely taught the underlying cause of conditions during their training. Instead they learn about treating the symptoms of disease that are treated with drugs.

The underlying cause of FM is excess adrenaline. The list provided of co-morbid conditions are basically all symptoms of excess adrenaline. So what I am proposing is that fibromyalgia be treated by dealing with the reason why the body is releasing excess adrenaline all day and night even though the person is not in danger.

It is actually easy to accomplish, and even though it is said that there is no cure for fibromyalgia, it can actually be eliminated in as little as three days to three weeks. I have written several books on how to do this that explains how to do this, especially my book called "Adrenaline Dominance".

Michael E. Platt, md
Reply Reply

Posted by: Gal@2016
May 3, 2017
Excess adrenaline could cause sleeplessness. I have 25 years of sleep disorder. 13 years of Fibro. I am using a sleep supplement and see improvement in sleep and Fibro symptoms.


Science uses buckshot
Posted by: Gal@2016
May 3, 2017
Focus. It is not so big. Science is using to broad focus and hoping to hit something.
Reply Reply

Immune System, Reactivates HSV-1 (Cold Sore Virus)
Posted by: CDW12
May 6, 2017
I believe the immune system is the root of this disease. Then HSV-1 (Cold Sore Virus) reactivates and impacts many body systems including the central nervous system.

Pridgen is embarking on Phase III clinical trials fast-tracked by the FDA. Although the immune system may be the actual disease issue, our science and healthcare are not able to work on the immune system with any great success and if a symptom (HSV-1) needs to be suppressed because the patient's immune system cannot do it then this should be the first step in helping patients.

I believe EpicGenetics is also on the right course but even if they prove out their tests are accurate for detection of the disease and it links back to genetics, we still need to work on the symptoms and IMC-1 (name of Pridgen's test drug) needs to be tested in Phase III. Off-label drugs will probably be used that make up his fixed dosage in IMC-1 but that is done with all new drugs.

See paragraph 3 of Introductory information for the 'Fibromyalgia' page on MEpedia.
Reply Reply

Sorry, Paragraph 4
Posted by: CDW12
May 6, 2017
Paragraph 4 of the 'Fibromyalgia' page on the MEpedia wiki.


No underlying cause
Posted by: IanH
May 15, 2017
There is NO underlying cause to FM.
There are many pathologies which are associated with the development of wide-spread pain, as the author above says.

Diagnostics confuses everyone because diagnosis is a subjective clinical decision.
Its like saying that headaches are caused by tension, some are and some are not.

There are clearly some genetic factors and some environmental factors. How these interact to produce chronic wide-spread pain we do not know.There are many different symptoms occurring in different people - this alone tells us we are not dealing with one cause or one "condition". These different "conditions" must be separated in order to understand the syndrome. There may be some common cellular abnormalities but I suspect there will be many of them.

This is why we need more decent research in FM - like what is happening in ME/CFS. I do suspect we will discover the underlying "causes" of FM via the research into ME/CFS because it is more thorough.
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