Journal: Med Hypotheses 2002 Mar;58(3):249-50
Authors: Tamizi far B, Tamizi B.
Affiliation: Department of Research, Isfahan University of Medical Sciences, Isfahan, Iran
NLM Citation: PMID: 12018979
Minor alterations of immune, neuroendocrine, and autonomic function may be associated with the chronic fatigue syndrome.
Omega-3 fatty acids decrease the production of putative mediators of inflammation, including interleukin-1, and tumor necrosis factor.
Since interleukin-1 and tumor necrosis factor are the principal polypeptide mediators of immunoregulation, reduced production of these cytokines by dietary supplementation with omega-3, may be a possible mechanism for the treatment of chronic fatigue syndrome.
Copyright 2002, Elsevier Science Ltd. All rights reserved.