Black Cumin May Improve Memory and Protect the Nervous System in Neurodegenerative Disease

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Editor’s Note: Parkinson’s, MS and some other neurodegenerative diseases have been thought to be caused, at least in part, by Lyme disease. Therefore, Nigella Sativa (or black cumin) may be a useful compound for protecting against the damaging neurodegenerative effects of Lyme.
 
A review of Neuropharmacology Effects of Nigella sativa (black cumin) and Its Main Component, Thymoquinone
 
Abstract
 
Neuropharmacology is the scientific study of drug effect on nervous system. In the last few years, different natural plants and their active constituents have been used in neurological therapy. The availability, lower price, and less toxic effects of herbal medicines compared with synthetic agents make them as simple and excellent choice in the treatment of nervous diseases.
 
Nigella sativa, which belongs to the botanical family of Ranunculaceae, is a widely used medicinal plant all over the world. In traditional and modern medicines several beneficial properties have been attributed to N. sativa and its main component, thymoquinone (TQ).
 
In this review, various studies in scientific databases regarding the neuropharmacological aspects of N. sativa and TQ have been introduced. Results of these studies showed that N. sativa and TQ have several properties including anticonvulsant, antidepressant, anxiolytic, anti-ischemic, analgesic, antipsychotic, and memory enhancer.
 
Furthermore, its protective effects against neurodegenerative diseases such as Alzheimer, Parkinson and multiple sclerosis have been discussed. Although there are many studies indicating the beneficial actions of this plant in nervous system, the number of research projects relating to the human reports is rare. Copyright © 2016 John Wiley & Sons, Ltd.
 
Source: By Javidi S1, Razavi BM2, Hosseinzadeh H3. A review of Neuropharmacology Effects of Nigella sativa and Its Main Component, Thymoquinone. Phytother Res. 2016 Aug;30(8):1219-29. doi: 10.1002/ptr.5634. Epub 2016 May 11.
 
 

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