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Chronic Fatigue Syndrome (CFS): an immunological perspective

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OBJECTIVE: The aim of this study is to review research

examining an immunological basis for chronic fatigue syndrome

(CFS) and to discuss how a disturbance in immunity could

produce central nervous system (CNS)-mediated symptoms.

METHOD: Data relevant to the hypothesis that abnormal cytokine

release plays a role in the pathogenesis of CFS are reviewed

as well as recent evidence relating to potential mechanisms by

which immune products may enter the brain and produce a

disturbance in CNS processes.

RESULTS: Examinations of

cytokine levels in patients with CFS have produced

inconclusive results. Recent evidence suggests that abnormal

release of cytokines within the CNS may cause neural

dysfunction by a variety of complex mechanisms.

CONCLUSION:

Neuropsychiatric symptoms in patients with CFS may be more

closely related to disordered cytokine production by glial

cells within the CNS than to circulating cytokines. This

possibility is discussed in the context of unresolved issues

in the pathogenesis of CFS.

1 Star2 Stars3 Stars4 Stars5 Stars (76 votes, average: 2.90 out of 5)
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