One single treatment that inhibited the protein PTB converted astrocytes into neurons that produce dopamine, leading to the disappearance of Parkinson's disease symptoms in mice.

The gene RBFOX1 drives the initial appearance of amyloid plaques in the brain. Targeting those with increased RBFOX1 expression may be able to halt the development of Alzheimer's disease.

After a heart attack, there is significant damage to the tissues, proteins, muscles, and oxygen flow surrounding the heart. The protein MANF reduces this damage and improves recovery and survival.

The rate at which genetic mutations accumulate predicts lifespan and the remaining female fertile years. Adults who accumulated fewer genetic mutations lived 5 years longer than those with more mutations.