Male baboons who climbed the social ladder faster and competed the most to achieve dominance in the group had faster rates of biological aging, as measured by DNA methylation.

Deleting the protein tau led to improved cognitive abilities and mitochondrial function in normally aged mice; Tau protein buildup plays a vital role in the development of Alzheimer's disease.

A novel biomarker tracks the efficacy of senolytics — drugs that eliminate senescent cells that have permanently stopped dividing but remain dysfunctional in the body and promote inflammation.

A protein called membrane type 1 matrix metalloproteinase interferes with the body's ability to clear LDL ("bad") cholesterol, increasing the risk of heart disease.